I did my PhD in developmental biology at Leeds University and subsequently carried out postdoctoral studies with Professor Bryan Sykes at the Institute of Molecular Medicine at Oxford University. These entailed a molecular genetic analysis of monogenic and polygenic diseases of the musculoskeletal system. I subsequently obtained a fellowship from the Arthritis Research Campaign and established a group at the Wellcome Trust Centre for Human Genetics. At that point my focus became the genetic analysis of osteoarthritis (OA) and in 2008 I moved to Newcastle as Professor of Musculoskeletal Research.
I am a member of the following professional societies:
My groups principal research focus is identifying and then characterising those genes that confer risk towards the development and progression of osteoarthritis (OA).
Through a combination of linkage and association analysis the group has identified several genes harbouring susceptibility alleles for OA, including the genes GDF5, BMP5, DIO2, COG5 and COL11A1.
Our efforts are directed towards comprehensive functional analysis of the associated variants within these genes, and in others that are emerging from ongoing scans.
Current Group Members:
I was the principal investigator and coordinator of arcOGEN, a UK consortium of 16 investigators from 9 UK universities, whose aim was to conduct a powerful genome-wide association scan on 8000 OA cases and 11000 controls (www.arcogen.org.uk/). This study was supported by Arthritis Research UK (www.arthritisresearchuk.org/) with a grant of £2.2 million.
The scan is complete and the data analysed. We identified 5 highly significant signals, which cross the threshold for genome-wide significance, and 3 signals that are very close to being genome-wide significant. Our findings have been submitted for publication and the arcOGEN resources will soon be made available to other investigators.
I also have an active interest in those monogenic diseases of the extracellular matrix that have secondary OA as a phenotypic component, partly because these can highlight potential candidate genes for our primary OA studies.
I have also established collaborations with clinicians and scientists investigating other common complex phenotypes, including developmental dysplasia of the hip and exceptional longevity.
Many of the loci that my group investigate harbour polymorphisms that influence gene expression and this has stimulated a new research area in my group, an analysis of the tissue-specific effects of cis polymorphism on allelic expression imbalance.
The technical expertise that my group employs includes:
My groups main effort for the next 5 years will be to complete the genetic mapping of OA susceptibility loci and to carry out detailed functional studies of the loci identified such that we can understand clearly how the associated variants influence OA disease risk. This information will then be applied to develop improved diagnostic and prognostic tools and to facilitate the development of new intervention strategies, including novel therapeutics.
I have considerable experience of postgraduate supervision and I currently supervise five PhD students; one who has just started her second year, three who have just started their third year, and one who has just completed his laboratory studies and is now writing up. I encourage undergraduates who have a keen interest in genetics and the functional analysis of disease loci to get in touch.
Principal investigator on over 20 grants, co-applicant on an additional 8. Total grant income to date > £15 million.